Sarcoidosis Remission and Aden Protocol

Chinese Secrets To Sarcoidosis Healing

The most important aspects of the program describes in the ebook are: It is research based and scientifically proven. The explanations are simple , adjusted to the Western view, specific and precise. It will allow you to discover and put a stop to the. habits, diet and lifestyle choices that are keeping. you sick and making your healing a mission impossible. It reveals all the culprits that I identified to be the. reason why people in China don't get sarcoidosis. My practices proved that once these aggravating agents are removed from your life, self-healing aided with herbs and remedies becomes simply inevitable. It reveals the remedies made from the herbal healing. agents and supplements that boost the self-healing and speed up the recovery. It will save you thousands of dollars. in drugs, doctor appointments and expensive surgeries. Read more here...

Chinese Secrets To Sarcoidosis Healing Summary


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Highly Recommended

Of all books related to the topic, I love reading this e-book because of its well-planned flow of content. Even a beginner like me can easily gain huge amount of knowledge in a short period.

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Sarcoidosis Freedom Cookbook

This e-book is a 143 pages long downloadable ebook with hundreds of delicious recipes. But these recipes are not only delicious but have another crucial feature they use only the ingredients that are carefully chosen to meet all the criteria that are critically important in our pursuit of health and sarcoidosis remission. This is how The Sarcoidosis Freedom Cookbook will change your life: You will never eat a meal that triggers your sarcoidosis again. And you very likely did it today. You will gently soothe your endocrine system and shift the ravaging chemical imbalance that is eating away your organs. Kick start the boost of self-healing chemicals that will repair your organs before it's too late. Enjoy delicious meals while knowing every second that you are doing good to your body and getting closer to remission. You won't have to think about where to start in your healing, you will have all the work done for you. When you wake up in the morning you'll feel light and positive, knowing that healing chemicals in your body are doing their work every second. You won't have to spend endless hours in front of your computer or buy nutrition books to know what is completely safe for you. Never again buy another book about diet and health, because you have it all right here and written just for your condition, not general and vague. Start your healing today, without any procrastination. Once again, feel that health and energy you so desperately pursue. Read more here...

Sarcoidosis Freedom Cookbook Summary

Contents: 143 Page Ebook
Author: Danielle May
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Case 11 Bilateral Hilar And Mediastinal Adenopathy From Sarcoidosis

Asbestos Exposure Cxr

CXR shows bilateral symmetrically enlarged hilar and mediastinal lymph nodes. CT (Fig. 11.2) confirms this finding, typical of sarcoidosis. The main differential diagnoses would be lymphoma and tuberculosis, but the lymphadenopathy would then be asymmetrical. Bronchoscopy and transbronchial lung biopsy are positive in 60 of cases, showing non-caseating granulomas and culture negative for tuberculosis and fungus. Blind endobronchial biopsies increase the yield by another 20 but the gold standard is mediastinoscopy. Incidence in people of African origin is ten times higher than in Caucasians.

Sarcoid And Maxillary Sinus

Sinonasal Wegener Granulomatosis

Fection, sarcoidosis, connective tissue diseases, and neoplastic lesions (basically lymphomas, metastases, and fibrous tumors) (Dalley 1999 Weber et al. 1999). In all these conditions, imaging findings should be carefully matched with clinical information and laboratory tests, as even sophisticated information provided by MR are, in itself, often insufficient to provide the diagnosis. A very limited number of studies in the literature focus on imaging findings of sinonasal sarcoidosis - reflecting the low incidence of this condition. CT and MR findings may consist, in moderate stages, of isolated mucosal thickenings, intrasinusal air-fluid levels, and hypertrophy of turbinates, impossible to differentiate from a mere chronic sinusitis (KrESPi et al. 1995). In advanced stages, submucosal granulomas and bone erosions may be observed, shifting the differential diagnosis towards Wegener granu-lomatosis and cocaine induced midline destructive lesions. MR signal pattern of sarcoid...

Sarcoidosis 6531

Sarcoidosis is a systemic non-caseating granulo-matous disease that mainly involves lungs (80-90 of all patients) (FErgiE et al. 1999) and the upper respiratory tract, including the nasal fossae and paranasal sinuses (Ktespi et al. 1995 DeShazo et al. 1999). Other extra-respiratory localizations are lymph nodes, liver, spleen, eye, skin, bones, brain, salivary and lacrimal glands (DeShazo et al. 1999 MAZZiotti et al. 2001). The etiology of sarcoidosis is still unknown. Some immunological alterations (i.e., depression of T-lym-phocyte function, elevated B-lymphocyte activity and serum levels) are frequently found in affected subjects and are hypothesized to play a relevant role in the onset of the disease. Also infective agents, such as atypical Mycobacteria, are listed among the potential etiologic factors (FErgiE et al. 1999). reports, however, confirmed sinonasal tract involvement in no more than 6 of all cases of sarcoidosis (Marks and GooDmAN et al. 1998 DAmRosE et al. 2000...

The Mediators Expressed by Mast Cells and Their Role in the Inflammatory Response

Mast cells have been incriminated in such diverse diseases as allergy, asthma, rheumatoid arthritis, atherosclerosis, interstitial cystitis, inflammatory bowel disease, progressive systemic sclerosis, chronic graft-vs-host disease, fibrotic diseases, sarcoidosis, asbestosis, ischemic heart disease, keloid scars, and malignancy (3). The mediators released by mast cells can independently and, in synergy with macrophage- and T-cell-derived cytokines, induce much of the inflammatory pathology observed in inflammation and serve to orchestrate a complex immune response. Histamine, LTB4, LTC4, PAF, and PGD2 may have multiple effects on inflammatory cell recruitment (eosinophils), smooth muscle hyperplasia, and vascular dilatation (80,81). Tryptase, chymase, and TNF-a from mast cells activate fibroblasts, leading to collagen deposition and fibrosis. Mast cell-derived TNF-a regulates NF-KB-dependent induction of endothelial adhesion molecule expression on endothelial cells in vivo (49). Mast...

Crystalinduced disease

Sarcoidosis presents as a periarthritis or polyarthritis associated with hilar adenopathy, erythema nodosum, and fever (Lofgren syndrome). A similar disorder can occur without adenopathy and can be due to infections (streptococci, TB, coccy), inflammatory bowel disease, and drug reactions.

Macrophages in immune response sites

Specific types of infections give rise to granulomatous inflammation. These include tuberculosis, sarcoidosis, cat-scratch disease, leprosy, brucellosis, and schistosomiasis (49, 50). A granuloma is identified by its distinctive pattern of inflammatory reaction in which aggregates of epithelial-like macrophages are surrounded by lymphocytes and a few plasma cells. Older granulomas also display an outer layer of fibroblasts and connective tissue. In some cases, fused macrophages, giant cells, are also found (49). Protocol 19 describes the isolation of granuloma macrophages (51) from livers or lungs (52) of infected mice.

Diffeentiall diagnosis Treatment

The clinical manifestations of the disease were first described in 1888. In 1933, Henrik Sjogren published a monograph describing in great detail the histologic and clinical components of this syndrome, which has often been referred to as Sjogren's syndrome. Confusion later arose between SS and the disease previously reported by Mikulicz in the late 1800s. In 1927, Schaffer and Jacobsen defined two main categories (a) Mikulicz's disease proper, of unknown etiology and following a benign course, and (b) Mikulicz's syndrome, caused by a variety of disorders such as leukemia, lymphosarcoma, tuberculosis, sarcoidosis, and iodide poisoning. Later, Morgan and Castleman concluded, on the basis of pathologic descriptions, that Mikulicz's disease and SS were identical. Much of the interest in SS during the past decade has focused on the opportunity to study the interrelationships among the autoimmune disorders, lymphoproliferative malignancies, and dysproteinemias, all of which may be features...

Isolated angiitis of the central nervous systen is a recently recognized vasculitic disorder primarily involving the

Patients usually have normochromic, normocytic anemia, leukocytosis, thrombocytosis, and an elevated ESR. WG, in contrast to the immune complex vasculitides, is associated with antineutrophil cytoplasmic antibody (ANCA), especially c-ANCA, which has a 98 specificity but a 30 to 99 sensitivity. Titers are usually associated with disease activity consequently, only 30 to 40 of patients with limited WG or generalized WG in remission have c-ANCA positivity. A small minority (5 ) can be positive for p-ANCA also. The limited form of the disease can be difficult to diagnose on clinical grounds, and the presence of c-ANCA may strongly influence the diagnosis. The strongest diagnostic evidence nonetheless comes from biopsy specimens of the involved tissues, which show granulomas. Lymphomatoid granulomatosis and necrotizing sarcoidosis may be confused with WG. The causative agent(s) leading to granuloma formation are unknown.

Case 17 Mycetoma Right Upper Lobe

Fungus Ball Spondilitis

The CXR shows a right upper lobe ball within a cavity (air crescent sign) patho-gmonic of a mycetoma (also called aspergilloma). A lateral decubitus X-ray may demonstrate the fungal ball shifting position. In this condition, a preformed cavity becomes colonized, usually by the fungus Aspergillus fumigatus. Cavitary disease may be secondary to fibrotic lung disease, e.g. previous tuberculosis, sarcoidosis, or ankylosing spondylitis. Massive hemoptysis can result and bronchial angiogram with embolotherapy (using coils or gel foam) is temporizing. Surgical resection is definitive, but bronchopleural fistula may result. Unfortunately, most patients have insufficient pulmonary reserve to allow surgical resection.

Case 61 Tracheal Stenosis Due To Tracheopathia Osteochondroplastica

Tracheopathia Osteochondroplastica

The CXR shows narrowing of the tracheal air column (Fig. 61.2) with calcification of the wall. Tracheal narrowing can be due to malignant causes (lung cancer, lymphoma, metastases) or benign causes (post tuberculosis, posttraumatic, amyloidosis, sarcoidosis, Wegener's, Tracheopathia Osteochondroplastica). Tracheopathia Osteochondroplastica (TO) is an extremely rare condition, characterized by the presence of multiple osseous and or cartilaginous submucosal nodules (Fig. 61.3) protruding into the lumen of the airway. Bronchoscopy is diagnostic but treatment is nonspecific and supportive.

Vt Related To Regions Of Scar

Scar Related Reentry

The most common cause of VT is reentry through regions of scar, most commonly an old MI (Fig. 4). Other scar-related VTs occur because of arrhythmogenic right ventricular dysplasia, sarcoidosis, Chagas' disease, and other nonischemic cardiomyopathies. Two features of ventricular scarring lead to reentrant VT (40-42). First, dense scarring creates regions of anatomic conduction block. Second, the scar is not comprised completely of dense fibrotic tissue, but also contains surviving myocyte bundles (43,44). Fibrosis between myocytes and myocyte bundles decreases cell-to-cell connections. The excitation wavefront propagates in a zig-zag manner from myocyte bundle to myocyte bundle, increasing the time for depolarization to procede through the region and thereby causing slow conduction (44). Circulation of an excitation wavefront around an area of block leads to reentry. With slow conduction, each cell in the circuit has sufficient time to recover after each depolarization.

Selective IgA Deficiency

The second patient group has reduced numbers of B cells and defects in the synthesis of IgG and IgM. These patients present with granulomatous disease resembling sarcoid. Granulomata may be found in the spleen, lymph nodes, lungs, skin, and brain. The histology is characteristic ofnoncaseating epithelial granuloma (Spickett et al., 1990).

Case 36 Mediastinal Lymphadenopathy Due To Lymphoma

Hilum Overlay Sign

The CXR shows asymmetric distortion of the mediastinal contour by markedly enlarged lymph nodes overlying the left hilum. This is described as the hilar overlay sign - the normal left pulmonary artery (Fig. 36.2) is seen through the mass (lying at the anterior mediastinum). Other differential diagnoses include chronic lymphocytic leukemia, sarcoidosis, Castleman's disease, and granulomatous disease like tuberculosis or histoplasmosis. The histology from mediastinoscopy in this patient showed Non Hodgkin's lymphoma.

Echo Speckled Appearance

Speckled Pattern Echocardiogram Amyloid

Restrictive cardiomyopathy may be idiopathic or secondary to infiltrative diseases such as amyloidosis, hemochromatosis, hypereosinophilic syndrome and Loeffler endocarditis, sarcoidosis, radiation toxicity, glycogen storage diseases, and Gaucher disease544 (see also Chap. 75). Typical 2D echocardiographic features of these diseases include (1) a diffuse increase of ventricular thickness in the absence of marked ventricular chamber dilation and (2) marked biatrial enlargement509,545-549 (Fig. 13-109). Systolic function is often modestly decreased. As with the other cardiomyopathies, these echocardiographic findings are nonspecific. Doppler

Endocrine myopathies

Muscle biopsies in small numbers of sarcoid patients without symptoms of muscle pain or weakness have revealed noncaseating granulomas typical of the disease. The occurrence of asymptomatic muscle involvement has clouded the issue of whether a true sarcoid myopathy exists. Nonetheless, there are sarcoid patients with symptomatic muscle involvement. Muscle pain and tenderness are most often seen in acute sarcoidosis with erythema nodosum. Symmetric proximal muscle weakness can be seen in chronic sarcoid (see Chapters. ).

Physiopathology of Hypogonadism

Aneurysms of the cavernous sinus, as well as infectious (bacterial or fungal abscesses, tuberculosis, and toxoplasmosis) and inflammatory processes (sarcoidosis, giant cell granuloma, lymphocytic hypophysitis, and histiocytosis X) can mimic a pituitary adenoma, causing mass effect and hypothalamic-hypophyseal endocrine dysfunction secondary to infiltration of normal tissue (91).

Affected Organs And Cell Types In Polyomavirusassociated Disease And Persistent Virus Infection

Pregnancy is the most common condition of altered immunocompetence that has been linked to polyomavirus activation (Coleman et al., 1980 Lecatsas et al., 1981). The onset of virus excretion is related to time of gestation, most often occurring late in the second and during the third trimester. Once established, excretion continues intermittently to term and might even extend into the postpartum period (Coleman et al., 1980 Gardner and Knowles, 1994). Serologic studies revealed that excretion in pregnant women is usually the result of virus activation of a persistent infection. Although activation of infection had no clinical significance (Arthur et al., 1989), women excreting polyomaviruses had more illness before and during pregnancy and may have underlying diseases such as diabetes and sarcoidosis (Gardner and Knowles, 1994). When BKV and JCV viruria were differentiated, BKV excretion rates of 15-25 were observed (Jin et al., 1993 Markowitz et al., 1991). The incidence of JCV...


Pyoderma gangrenosum is frequently associated with ulcerative colitis, Crohn's disease, RA, myeloproliferative disorders, and leukemia. Rarely, it is associated with chronic active hepatitis, myeloma, sarcoidosis, and diabetes mellitus. It begins as a tender pustule that rapidly expands to become a large ulcer many centimeters in diameter with a bluish, undermined border and a necrotic, purulent center. Lesions most frequently occur on the legs and trunk and heal with scar formation. Cutaneous trauma may exacerbate existing ulcers or cause the formation of new lesions.

Cutaneous lesions

All cutaneous manifestations of sarcoidosis, except EN and transient maculopapular eruptions, show histologic evidence of sarcoid granulomas on skin biopsy. A. Erythema nodosum is a septal panniculitis that appears as painful, erythematous, slightly raised, rounded lesions symmetrically distributed on the extensor aspect of the extremities, often localized to the shins. Fever and polyarthralgias may occur. When healing, the lesions assume the color of a bruise. Recurrent lesions appear in crops. EN is not specific for sarcoidosis, but the condition is termed Lofgren syndrome when accompanied by bilateral hilar adenopathy. C. Granulomatous cutaneous lesions of sarcoidosis include the following


The diagnosis of LM in general can be difficult to make, since patients may initially have vague complaints and minimal neurologic findings. LM may go on to mimic other disorders, such as tuberculosis or fungal meningitis, CNS sarcoidosis or pseudotumor cerebri.10'31'76 As mentioned in the introduction of this chapter, lymphomatous meningitis and PDLG, primary neoplastic diseases of the leptomeninges, would present a similar picture. The diagnosis of LM for brain tumor patients can also be hampered by the vagueness of the symptoms that may be present, especially since the CNS is already involved with tumor. Occasionally, patients with primary

Surgical Training

The training of rhinologists should include the holistic management of patients. This should include not only training in how to communicate with patients but the medical management of patients whose disease is not solely confined to the nose (e.g., asthma, immunosuppression, sarcoid, vasculitis, and ciliary dysmotility). Many rhinologists are aware of the psychological aspects of rhinological symptoms (Homer et al., 2000), yet this subject is not well covered in the medical literature or in teaching and research. Many patients who complain bitterly of postnasal drip or catarrh as primary symptoms describe them as having a much greater effect on their life, or ability to function, than might be expected. One pervading theme for the future is that surgeons should not perceive themselves as being the only people who can help their patients and that a team approach (nurse practitioners, asthma and allergy nurses) involving other disciplines (immunologists, respiratory physicians,...

The Clinical Problem

Cytomegalovirus, HTLV-1, others Fungal (Candida, Aspergillus, others) Non-infectious Involving eye alone Pars planitis Idiopathic vitritis Idiopathic retinal vasculitis Idiopathic multifocal retinochoroiditis Sympathetic ophthalmia White dot syndromes Histoplasmosis-like disease Associated with systemic disease Behfet's disease Sarcoidosis

Case 9 Silicosis

Hilar Eggshell Calcifications

The CXR shows bilateral infiltrates and calcified nodules in both upper lobes. Differential diagnoses of upper lobe infiltrates include silicosis, tuberculosis, and ankylosing spondylitis. There is also egg-shell calcification of the hilar lymph nodes. The egg-shell calcification plus the upper lobe nodules are typical of silicosis. Differential diagnoses of egg-shell calcification include sarcoidosis, Hodgkin's lymphoma following radiotherapy, and coal-worker's pneumoconiosis.

Nasolacrimal Duct

Nasolacrimal Duct Obstruction Scan

Teration in refraction that requires the patient to repeatedly blink or wipe their eye. Its prevalence is much more common with aging. It is unusual for intranasal pathology to be responsible, but conditions such as Wegener granulomatosis and sarcoidosis can affect this (Fig. 14.3 a). Nasolacrimal duct obstruction can occur

Otitis Externa

Otitis externa is generally caused by organisms such as Staphylococcus aureus and Pseudomonas aeruginosa. Malignant otitis externa is almost always due to P. aeruginosa (see Table 1). Only rare cases of malignant otitis externa due to S. aureus (6), Proteus mirabilis (7), and Aspergillus fumigatus (8) have been reported. Rare causes of chronic otitis externa include tuberculosis, fungal infections, syphilis, yaws, leprosy, and sarcoidosis. Fungal otitis externa may be part of a general or local fungal infection Aspergillus spp. are responsible for the most cases (9).

Prader Willi Syndrome

Structural lesions of the hypothalamus and pituitary can lead to an abnormal pattern of GnRH and or gonadotropin secretion (see Chapter 8). Most patients with such tumors have multiple pituitary hormone deficiencies, whereas isolated hypogo-nadotropism is rare (119,120). However, patients with mass lesions can present with hypogonadotropic hypogonadism without adrenal or thyroid hormone deficiency, but GH deficiency with or without hyperprolactimemia is almost always present. Indeed, a mass in the pituitary or hypothalamus is more likely to decrease the secretion of gonadotropins than that of adrenocorticotropic hormone (ACTH) or TSH. Cranio-pharyngioma is the most common tumor resulting in hypogonadotropic hypogo-nadism in childhood. It is often associated with growth retardation, diabetes insipidus, and visual field defects. Prolactinoma is the most frequent tumor causing hypogonadotropic hypogonadism in adult men (121,122). Although rare, infiltrative hypothalamus or pituitary...

U3 U4 U5 U6

Fig. 9.12-lead ECG recording of baseline (left) and VT (right) from a patient with cardiac sarcoidosis. Sarcoid VT frequently originates from the basal left ventricle, producing a RBBB VT morphology with positive concordance across the precordial leads. Sarcoidosis. Sarcoidosis is a chronic, multisystem disorder of unknown etiology characterized by noncaseating granulomas in affected organs. Although 5 of patients with sarcoid have symptomatic cardiac involvement, approx 20 have evidence of cardiac sarcoid at autopsy (50). Cardiac involvement is manifested by myocardial infiltration, CHF, heart block, VT (Fig. 9), and sudden death. Sarcoid granulomas preferentially affect the basal interventricular septum and left ventricular papillary muscles. The right and left ventricular free walls can also be involved. The resting ECG is nonspecific, but frequently abnormal. The mechanism of VT is unclear but ICD therapy has been advocated because sarcoid-related VT is often difficult to control...

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