Treating gum disease with homemade remedies

Freedom From Dental Disease

The Primary Care Oral Health Action Pack can Favorably change the way you look at your oral health and what affects it. Increase your knowledge of YOUR teeth and gums, YOUR entire oral environment and yes, your SMILE! Reveal the TRUE CAUSE of cavities, decay of bone and tissue and disclose the proper biological balance. Put YOU in command over this contagious, yet EASILY preventable disease which runs rampant thoughout our population. Allow you to TAKE ACTION to restoring your teeth and gums to optimum health while preventing further decay and damage, as well as preventing any potentially related problems such as heart disease, diabetes, and so on. How to Become Dentally Self Sufficient. Research Advocates OraMedics The science behind the program! The 7 Factors Transcript - Dr. Nara's Last speech before the World Health Federation. Read more...

Freedom From Dental Disease Summary


4.6 stars out of 11 votes

Format: Ebook
Official Website:
Price: $19.95

Access Now

My Freedom From Dental Disease Review

Highly Recommended

The very first point I want to make certain that Freedom From Dental Disease definitely offers the greatest results.

As a whole, this book contains everything you need to know about this subject. I would recommend it as a guide for beginners as well as experts and everyone in between.

What You Should Know About Gum Disease

These books encapsulate my personal experience with fighting gum disease, described as follows: My gums bled during dental cleanings. I had pockets depths of 4 and 5mms and one 6mm pocket. My dentist told me I had moderate gum (periodontal) disease. She and / or the hygienist also told me that the ONLY thing that would help me was a Scaling and Root Planing Treatment - Otherwise knowns as a SRP or Deep Cleaning. After applying the information I learned on my own and the tools that I employed the results were that my pocket depths returned to normal. There was no more bleeding during dental cleanings, brushing or flossing. I was told that I no longer needed the Deep Cleaning or SRP treatment and that whatever I was doing, I should keep it up. Read more...

What You Should Know About Gum Disease Summary

Format: Ebook
Official Website:
Price: $19.65

Leukocyte Adhesion Deficiency I

Infection often progressively enlarge, and they may lead to systemic spread of the bacteria. Infections are usually apparent from birth onward, and a common presenting infection is omphalitis with delayed separation of the umbilical cord. The most frequently encountered bacteria are Staphylococcus aureus and gram-negative enteric organisms, but fungal infections are also common. The absence of pus formation at the sites of infection is one of the hallmarks of LAD I. Severe gingivitis and periodontitis are major features among all patients who survive infancy. Impaired healing of traumatic or surgical wounds is also characteristic of this syndrome (Anderson and Smith 2001).

Gerontologie Alert

Long-term administration of the hydantoins can cause gingivitis and gingival hyperplasia (overgrowth of gum tissue). It is important to periodically inspect the teeth and gums of patients in a hospital or long-term clinical setting who are receiving one of these drugs. The nurse reports any changes in the gums or teeth to the primary health care provider. It is important that oral care be given after each meal and that the mouth and gums be inspected routinely.

Extraintestinal Infection

Secondary complications resulting from primary Campylobacter enteritis are relatively rare. These complications include cholecysitis, pancreatitis, peridontitis, gastrointestinal hemorrhage, meningitis, endocarditis, anthritis, peritonitis, cellulitis, hepatitis, and septic abortion. With improved methods for identification of Campylobacter, more species are now recognized to be important in causing human infections, especially when they infect immunocompromised persons. For example, C. fetus subsp. fetus is primarily associated with infectious abortion in cattle and sheep and is an infrequent cause of human infections. C. fetus can cause fetal death or septicemia in debilitated persons with chronic disease or in immunosuppressed patients (Blaser, 2000). C. jejuni and C. coli can also cause human abortion. Other Campylobacter spp., C. concisus, C. rectus, and C. curvus are known to be implicated in human periodontal disease (Tanner et al., 1987).

Chronic Granulomatous Disease

CGD is a genetic disease that has at least two distinct forms an X-linked form that occurs in about 70 of patients and an autosomal recessive form found in the rest. This disease is rooted in a defect in the oxidative pathway by which phagocytes generate hydrogen peroxide and the resulting reactive products, such as hypochlorous acid, that kill phagocytosed bacteria. CGD sufferers undergo excessive inflammatory reactions that result in gingivitis, swollen lymph nodes, and nonmalignant granulomas (lumpy subcutaneous cell masses) they are also susceptible to bacterial and fungal infection. CGD patients are not subject to infection by those bacteria, such as pneumococcus, that generate their own hydrogen peroxide. In this case, the myeloperoxidase in the host cell can use the bacterial hydrogen peroxide to generate enough hypochlorous acid to thwart infection. Several related defects may lead to CGD these include a missing or defective cytochrome (cyt b55s) that functions in an oxidative...

Multiple Intracellular Microbial Strategies

During the invasion of macrophages by a microorganism, both the host and the pathogen may elicit mechanisms that alter iron metabolism within the macrophage. The interested reader is referred to a recent elegant review on this topic by Eugene Weinberg, who for decades has pioneered research on iron and infection (Weinberg, 2000). One of the major weapons utilized by the macrophage against intracellular parasitism is associated with its stimulation by g-interferon. This T-cell derived cytokine promotes several activities that are relevant to iron metabolism. First, it induces a decreased expression of the number of transferrin receptors at the surface of the macrophage, resulting in a limited cellular iron uptake and impaired iron acquisition by microorganisms such as Legionella pneumophila that escapes into the cytosol and are likely to be dependent on the LIP for growth (Byrd and Horwitz, 1989). The same authors recently reported an 'experiment of nature', namely a patient, whose...

Clinical Features

When the CD44 T-cell count falls below about 400 per (xl the patient may develop a constellation of constitutional symptoms (fever, night sweats, oral candidiasis, diarrhea, and weight loss) which used to be known as AIDS-related complex (ARC). Early opportunistic infections begin to be seen. At this intermediate stage of immune depletion these infections are generally not life-threatening. They particularly include infections of the skin and mucous membranes such as tinea, seborrheic dermatitis, bacterial folliculitis, warts, molluscum contagiosum, gingivitis, oral and esophageal candidiasis, oral hairy leukoplakia (Fig. 35-10D), and chronic sinusitis. Reactivation of latent herpesviruses, particularly herpes simplex and zoster, also occurs (see Chapter 20). Gastrointestinal infections, caused by any of a wide vanety of organisms, including the yeast Candida albicans and parasites such as Cryptosporidia, are common. Mycobacterial infections are also common in these patients, and this...

Leukocyte Adhesion Deficiency II

Affected children were born after uneventful pregnancies with normal height and weight. No delay in the separation of the umbilical cord was observed. They have severe mental retardation, short stature, a distinctive facial appearance, and the rare Bombay (hh) blood phenotype. From early life, they have suffered from recurrent episodes of bacterial infections, mainly pneumonia, periodontitis, otitis media, and localized cellulitis. During times of infections, the neutrophil count increases up to 150,000 l. Several mild to moderate skin infections, without obvious pus have also been observed (Wild et al. 2002). The infections have not been life-threatening events and are usually treated in the outpatient clinic. Interestingly, after the age of 3 years, the frequency of infections has decreased and the children no longer need prophylactic antibiotics. At older age, their main infectious problem is severe perio-dontitis as is also observed in patients with LAD I (Etzioni et al. 1998)....

Evaluation of Suspected Immunodeficiency

Dihydrorhodamine Test

Abnormalities in Toll-like receptor (TLR) function recently has been defined in patients with a specific pattern of bacterial infections (Ku et al. 2005). These findings suggest that additional innate immune defects impacting this early response pathway are likely. Innate immune defects affecting neutrophil function typically result in cutaneous and deep-seated abscesses, pneumonia, periodontitis, and osteomyelitis (Rosenzweig and Holland 2004). These infections are often caused by characteristic bacteria (e.g., Staphylococcus aureus and Serratia marcesens) and or fungi (e.g., Aspergillus and Nocardia species). Congenital defects in specific complement components often are associated with autoimmunity as well as recurrent bacterial infections similar to the antibody deficiencies (Walport 2001). In addition, abnormalities of the late complement components are associated with a unique increase in susceptibility to Neiserrial infections (Figueroa and Densen 1991). with a Boyden chamber...

Clinical Manifestations 21 Orofacial Infections

Infection of the pulp can occur in one of three ways (1) through a defect in the enamel and dentine due to extension of a carious lesion, traumatic fracture, or a dental procedure (2) through the apical foramen or lateral canals (e.g., from a periodontal pocket or an adjacent tooth with a periapical abscess) and (3) through hematogenous seeding of the pulp that has been irritated mechanically. Once infected, the acute inflammatory reaction causes a rapid accumulation of pressure inside this rigid and unyielding space, compressing the blood vessels entering the pulp cavity through the apical foramen, and causing ischemia and necrosis of the pulp tissue. Pus may egress out of a cavity in the crown if one exists or may extrude apically into the surrounding periodontal tissue resulting in an acute periapical periodontitis. Alternately, the infected material may erode out of the apical foramen resulting in a periapical or alveolar abscess. The accumulation of pus causes loss of bone and...

Epidemiology And Clinical Relevance

Orofacial and odontogenic infections are diverse in etiology and clinical presentation. Elderly patients are particularly at risk because of poor oral health and relatively high prevalence of dental caries and periodontal disease. Such infections in the elderly may be localized and indolent, or invasive and life-threatening. Patients with systemic underlying diseases such as diabetes mellitus are also prone to more serious infections. The increasing need for valvular and joint replacements in the elderly also exposes this population to a greater risk for serious complications such as infective endocarditis or prosthetic infections from hematogenous seeding of odontogenic infections (1). 1.1. Prevalence of Dental Caries and Periodontal Disease in the Elderly Longitudinal studies of oral health in the aging person are generally rare. Nordstrom and colleagues (7) conducted a 9-yr longitudinal study of dental and periodontal status in 70- and 79-yr-old city cohorts in northern Sweden. The...

What features should be considered in the extraoral examination

The clinical examination should be directed to gaining specific evidence. In the previous chapter general aspects have been considered and emphasis has been placed on selecting those patients with a well-restored stable occlusion and a high standard of plaque control associated with non-progressive periodontal disease.


Inflammation of the tissues around an endosseous implant is sometimes observed it is described as peri-implant mucositis when it involves only the soft tissues and peri-implantitis where loss of the bone interface occurs. While the microorganisms associated with these lesions are similar to those seen in periodontal disease, it is currently unclear whether they cause the lesion or colonize the region subsequently.

The gums

At first bleeding is apt to occur, and a narrow line of inflammation can be seen at the free border of the gum, and the interdental papillae are swollen. If the condition progresses, food debris, bacteria and pus tend to accumulate between the teeth and the gum margin (pyorrhoea alveolaris). Halitosis may be apparent and the leelh may become loose. Pus or even a tooth may be aspirated into the bronchial tree and initiate pneumonia, badly affected gums are associated with frequent transient bacteraemia, the usual organism being Streptococcus viridans. This may cause infective endocarditis, especially in patients with valvular heart disease. Painful ulcere-membranous gingivitis may be due to Vincent's infection (p. 56)

Diagnostic Tests

An orthopantomogram may reveal the true extent of advanced periodontitis or the presence of periapical abscess. Ultrasonography, radionuclide scanning, computed tomography (CT), and magnetic resonance imaging (MRI) are particularly useful for the localization of deep fascial space infections of the head and neck. A lateral radiograph of the neck may demonstrate compression or deviation of the tracheal air column or the presence of gas within necrotic soft tissues. In retropharyngeal infections, lateral radiographs of the cervical spine or CT scanning can help determine if the infection is in the retropharyngeal space or the prevertebral space. The former suggests an odontogenic source, whereas the latter suggests involvement of the cervical spine. Technetium bone scanning, used in combination with gallium- or indium-labeled white blood cells, is particularly useful for the diagnosis of acute or chronic osteomyelitis and for the differentiation of infection or trauma from malignancy.


Dental Caries and Periodontitis With the improvement of dental restorative materials such as bonding and fluoride-releasing agents, dental caries in older adults can be readily treated by restorative and prosthodontic treatment programs. Acute necrotizing ulcerative gingivitis should be treated with systemic antimicrobials such as metronidazole or penicillin. Certain types of severe periodontitis are amenable to systemic antimicrobials in conjunction with mechanical debridement (scaling and root planing) (20). Double-blind clinical studies of advanced periodontitis in which systemic metronidazole (500 mg three times day orally) or doxycycline (200 mg two times d orally) for 1 or 2 wk used in conjunction with rigorous mechanical debridement was found to reduce the need for radical surgery by 80 compared with debridement plus placebo control. Oral antimicrobial rinses with 0.12 chlorhexidine is useful for the control of dental plaque bacteria that leads to gingivitis and...


The risk of infection is inversely proportional to the ANC. When the ANC falls below 1000 mm3, stomatitis, gingivitis, and cellulitis dominate the clinical picture. More severe infections occur when the ANC is below 500 mm3 with perirectal abscesses, pneumonia, and sepsis being common.

Download Instructions for Freedom From Dental Disease

You can safely download your risk free copy of Freedom From Dental Disease from the special discount link below.

Download Now